Journal Articles
Hatch, Robert John, Wei, Yan, Xia, Di and Gotz, Juergen (2017) Hyperphosphorylated tau causes reduced hippocampal CA1 excitability by relocating the axon initial segment. Acta Neuropathologica, 1-14. doi:10.1007/s00401-017-1674-1
Chai, Ye Jin, Sierecki, Emma, Tomatis, Vanesa M., Gormal, Rachel S., Giles, Nichole, Morrow, Isabel C., Xia, Di, Götz, Jürgen, Parton, Robert G., Collins, Brett M., Gambin, Yann and Meunier, Frédéric A. (2016) Munc18-1 is a molecular chaperone for α-synuclein, controlling its self-replicating aggregation. The Journal of Cell Biology, 214 6: 705-718. doi:10.1083/jcb.201512016
Xia, Di, Gutmann, Julia M. and Gotz, Jurgen (2016) Mobility and subcellular localization of endogenous, gene-edited Tau differs from that of over-expressed human wild-type and P301L mutant Tau. Scientific Reports, 6 . doi:10.1038/srep29074
Xia, Di, Li, Chuanzhou and Gotz, Jurgen (2015) Pseudophosphorylation of Tau at distinct epitopes or the presence of the P301L mutation targets the microtubule-associated protein Tau to dendritic spines. Biochimica et Biophysica Acta - Molecular Basis of Disease, 1852 5: 913-924. doi:10.1016/j.bbadis.2014.12.017
Xia, Di and Gotz, Jurgen (2014) Premature lethality, hyperactivity, and aberrant phosphorylation in transgenic mice expressing a constitutively active form of Fyn. Frontiers in Molecular Neuroscience, 7 MAY: 1-11. doi:10.3389/fnmol.2014.00040
Goetz, Juergen, Xia, Di, Leinenga, Gerhard, Chew, Yee Lian and Nicholas, Hannah R. (2013) What renders TAU toxic. Frontiers in Neurology, 4 72.1-72.10. doi:10.3389/fneur.2013.00072
Thesis
Xia, Di (2017). The molecular mechanism of the Tau-Fyn interaction and dendritic targeting in neurons PhD Thesis, Queensland Brain Institute, The University of Queensland. doi:10.14264/uql.2017.751