Dr Richard Lucas
Postdoctoral Research Fellow
Institute for Molecular Bioscience
+61 7 334 62035
Book Chapter
Tong, Samuel J., Lucas, Richard M., Xiao, Zhijian, Luo, Lin and Stow, Jennifer L. (2021). Detecting Endogenous Rab8 Activation. Rab GTPases: Methods and Protocols. (pp. 45-56) New York, NY United States: Humana Press. doi: 10.1007/978-1-0716-1346-7_4
Journal Articles
Liu, Liping, Lucas, Richard M., Nanson, Jeffrey D., Li, Yan, Whitfield, Jason, Curson, James E.B., Tuladhar, Neeraj, Alexandrov, Kirill, Mobli, Mehdi, Sweet, Matthew J., Kobe, Bostjan, Stow, Jennifer and Luo, Lin (2022). The transmembrane adaptor SCIMP recruits tyrosine kinase Syk to phosphorylate Toll-like receptors to mediate selective inflammatory outputs. Journal of Biological Chemistry, 298 (5) 101857, 101857. doi: 10.1016/j.jbc.2022.101857
Lucas, Richard M., Liu, Liping, Curson, James E.B., Koh, Yvette W.H., Tuladhar, Neeraj, Condon, Nicholas D., Das Gupta, Kaustav, Burgener, Sabrina S., Schroder, Kate, Ingley, Evan, Sweet, Matthew J., Stow, Jennifer L. and Luo, Lin (2021). SCIMP is a spatiotemporal transmembrane scaffold for Erk1/2 to direct pro-inflammatory signaling in TLR-activated macrophages. Cell Reports, 36 (10) 109662, 1-20. doi: 10.1016/j.celrep.2021.109662
Luo, Lin, Lucas, Richard M., Liu, Liping and Stow, Jennifer L. (2019). Signalling, sorting and scaffolding adaptors for Toll-like receptors. Journal of Cell Science, 133 (5), jcs239194. doi: 10.1242/jcs.239194
Luo, Lin, Curson, James E. B., Liu, Liping, Wall, Adam A., Tuladhar, Neeraj, Lucas, Richard M., Sweet, Matthew J. and Stow, Jennifer L. (2019). SCIMP is a universal Toll‐like receptor adaptor in macrophages. Journal of Leukocyte Biology, 107 (2) JLB.2MA0819-138RR, 251-262. doi: 10.1002/jlb.2ma0819-138rr
Preprint
Lucas, Richard M., Bauer, Claudia S., Chinnaiya, Kavitha, Schwartzentruber, Aurelie, MacDonald, Ruby, Collins, Mark O., Aasly, Jan O., Bronstad, Gunnar, Ferraiuolo, Laura, Mortiboys, Heather and De Vos, Kurt (2019). LRRK2-mediated phosphorylation of HDAC6 regulates HDAC6-cytoplasmic dynein interaction and aggresome formation.